
Gout is chief among the brutal inflammatory health conditions that a person may suffer. It manifests mainly on the big toe, but it may affect other joints as well. With gout, the affected joint becomes tender, swells, is painful, may reveal some redness, and feel warm or hot. Gout is prevalent, and many keep asking the question, “can you die from gout?” We will help you answer that question. Just read on.
Can you die from gout?
Yes and no. The yes applies where having gout increases the risk of death from the chronic risk factors or health conditions that may develop. The no applies whereby, if you suffer from gout and keep it under control through medication, it can be treated, and you won’t die from it. Gout will not directly kill you. However, it can lead to serious health problems that increase your risk of dying. Choosing to leave your gout untreated and not focusing on lowering the bloodstream’s uric acid levels also raises your death risk.
Gout and premature deaths
Studies have found that those with gout are at a higher risk of premature death than those that do not. Causes of gout play a significant role in mortality rates, and the occurrence of gout is linked to premature deaths. Why? You may ask. Gout is associated with the development of other health conditions that can cause eventual death.
Gout is associated with metabolic syndrome – a combination of metabolic and cardiovascular health risk factors including hypertension, diabetes, renal failure. Having these conditions puts you at a higher risk of death. These conditions are chronic and will all somehow be connected. They mainly occur alongside each other. Such a combination makes these conditions even more dangerous because they increase the risk of death.
If you have gout, you are at more significant risk for a heart attack or stroke. You can also suffer from insulin resistance, whereby there is a deterioration in the ability of the body to use insulin to reduce blood sugar. Hypertension can damage blood vessels in the kidneys and eventually cause renal failure. A diabetic patient is likely to suffer from high cholesterol levels and high blood pressure. This can bring forth cardiovascular conditions. Having uncontrolled high blood pressure raises the risk for a heart attack.
Untreated, chronic, or severe gout is a death risk
Mostly, gout is treatable with medications. However, the risk of death increases if it goes untreated to become chronic. This is usually evident, especially for those that take this inflammatory condition lightly. Some may view it as a simple intermittent episode of joint pain. The truth is that inflammation is always “busy” on the joints for gout patients. Inflammation keeps happening even when symptoms of acute gout improve. Simply having a chronic condition increases your risk for an earlier death. Those with gout need to receive urate-lowering medicine. However, only a small number of people get the treatment, and fewer stay on the medication consistently or long enough. Due to this, the uric acid crystals keep triggering the underlying and chronic inflammation.
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– Febuxostat For Gout – Is It A Valuable Remedy?
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– Aspirin And Gout – Is Aspirin A Good Pain Reliever For Gout?

Gout and serum uric acid levels
Critical to improving a gout patient’s survival rate is reaching the recommended serum urate target- 3.4-7.0 for males and 2.4-6.0 mg/dl for females. Having more than the recommended level puts you at a greater risk of death. Due to that, medications to lower higher urate levels must be administered. Some previous studies reveal that the inability to reach the targeted serum urate level is independently a predictor of gout patient mortality.
Suggested articles:
– Gout in Neck – Can you Get Gout in Your Neck?
– Is Gout Contagious? – Everything You Need to Know
– Gout And Genetics – Diet Or Genetics?
– Gout in Dogs – Can Dogs Get Gout?
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– Gout and Sleep Apnea – Is Gout Related to Sleep Apnea?
– Gout And Erectile Dysfunction – Can Gout Cause Erectile Dysfunction?
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The extreme pain of a gout flare is an alarm signal that something in the body is seriously wrong. It is up to the doctor to figure out the underlying cause, as recommended by Terkeltaub and Edwards in their book about gout. In a small minority of cases it is the warning of lead toxicity. In a large majority of cases it is the warning of chronic intermittent hypoxemia from sleep apnea, which is why most gout flares originate during sleep. The hypoxemia leads to gout by: generation of much excess uric acid from all oxygen-starved cells; serum acidosis which makes serum uric acid more likely to precipitate; increase in serum lactate, which leads to URAT1 reducing renal reabsorption of uric acid; and gradual reduction of the glomerular filtration rate which slows the removal of serum uric acid.
One of the first things that should be done when gout first occurs is a sleep study, followed by effective sleep apnea treatment when warranted, after which the hypoxemic causes of gout flares will no longer occur. If there is no investigation into the cause of gout, prescribing NSAIDs just muffles the alarm, and prescribing urate lowering drugs just disables the alarm. If the sleep apnea goes unresolved for too long, the patient’s risk will be significantly elevated for developing the many life-threatening consequences of sleep apnea, which usually are misunderstood to be metabolic comorbidities of gout, and lead to a shortened lifespan, even with the use of urate lowering drugs. Gout is an early warning!
Resolving the sleep apnea should result in no more gout flares. Urate lowering drugs will then probably be needed to accelerate the dissolution of all the urate crystals throughout the body that accumulated from all the previous apneic episodes. More information is available on YouTube, titled Gout is an Early Warning of Sleep Apnea.